Age-related macular degeneration : a comprehensive textbook by D. Virgil Alfaro III MD, Peter E. Liggett MD, William F.

By D. Virgil Alfaro III MD, Peter E. Liggett MD, William F. Mieler MD, Hugo Quiroz-Mercado MD, Rama D. Jager MD MBA FACS, Yasuo Tano MD

A popular crew of retina surgeons provides promising new advancements in age-related macular degeneration, with emphasis at the most important advances some time past 5 years. content material addresses all facets of administration, together with anatomy, body structure, and pathophysiology; imaging of the macula in age-related macular degeneration; prevention measures and therapy; remedy; and destiny instructions. Abundantly illustrated and fantastically prepared, this e-book serves as an exceptional reference and textbook.

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Tabatabay CA, D'Amico DJ, Hanninen LA, et al. Experimental drusen formation induced by intravitreal aminoglycoside injection. Arch Ophthalmol. 1987;105:826-830. 86. Lullmann-Rauch R. Experimentally induced lipidosis in cat retinal pigment epithelium. Graefes Arch Clin Exp Ophthalmol. 1981;215:297-303. 56. Miller JW, Adamis AP, Shima DT, et al. Vascular endothelial growth factor/vascular permeability factor is temporally and spatially correlated with ocular angiogenesis in a primate model. Am J Pathol.

Experimental subretinal neovascularization. ARVO abstracts. Invest Ophthalmol Vis Sci. 1993;34:1,168. 79. Gerschman R, Nadig PW, Snell AC, et al. Effect of high oxygen concentrations on eyes of newborn mice. Am J Physiol. 1954;179:115-118. 50. Kliffen M, Lutgens E, Daemen MJA, et al. The Apo*E3-Leiden mouse as an animal model for basal laminar deposit. Br J Ophthalmol. 2000;84: 1,415-1,419. 80. Smith LEH, Wesolowski, E, McLellan A, et al. Oxygen-induced retinopathy in the mouse. Invest Ophthalmol Vis Sci.

All of these factors strongly suggest integral involvement of inflammatory cells in the development of CNV. Finally, ischemia-based theories do not adequately explain the typical later stages of CNV in ARMD: the formation of scarring and regression of vessels. With time, the neovascularization appears to “burn out,” leaving a cicatricial mass almost completely devoid of vessels. If ischemia is the only cause for the vessels to grow, then once the CNV does grow, the capillaries of the CNV recapitulate the anatomy of choriocapillaris and overlying neurosensory retina.

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